[1]Morrison SJ, Scadden DT. The bone marrow niche for hematopoietic etem cell [J]. Nature, 2014, 505 (7483): 327-334.
[2]Ema H, Suda T. Two anatomically distinct niches regulates stem cell activity [J]. Blood, 2012, 120(11): 2174-2181.
[3]June Li. Quiescence regulators for hematopoietic etem cell [J]. Exp Hematol, 2011, 39(5): 511-520.
[4]Montecino-Rodriquez E, Leathers H, Dorshkid K. Expression of connexin 43 (Cx43) is critical for normal hematopoiesis [J].Blood,2000, 96(3):917-924.
[5]Jing PW, Hu MX, Song XY, et al. Characteristics of bone marrow stromal cells biology in aging rats model [J]. Acta Anatomica Sinica, 2015, 46(1): 44-50. (in Chinese)
景鹏伟,胡文煦,宋小英,等. 衰老大鼠模型骨髓基质细胞的生物学特点[J]. 解剖学报, 2015, 46(1): 44-50.
[6]Dai DF, Rabinovitch PS, Ungvari Z. Mitochondria and cardiovascular aging [J].Circ Res, 2012, 110(8): 1109.
[7]Velarde MC, Flynn JM, Day NU, et al. Mitochondria oxidative stress caused by Sod 2 deficiency promotes cellular senescence and aging phenotypes in the skin[J]. Aging (Albany NY ), 2012,4(1): 3-12.
[8]Sinon HU,Haj-Yehia A,Levi-Schaffer F. Role of reactive oxygen species (ROS) in apoptosis induction [J]. Apoptosis, 2000, 5(5):415-418.
[9]Taniguchi Isgukawa E, GonzalezNieto D, Ghiaur G, et al. Connexin-43 prevent hematopoietic stem cell senescence through transfer of reactive oxygen species to bone marrow stromal cells[J]. Proc Natl Acad Sci USA, 2012,109(23): 9071-9076.
[10]Schajnovitz A, Itkin T, D’uva G, et al.CXCL12 secrtion by bone marrow stromal cells is dependent on cell contact and mediated by connexin-43 and connexin-45 gap junctions[J]. Nat Immunol,2011, 12(5):391-398.
[11]Presley CA, Lee AW,Kasti B, et al. Bone marrow connexin-43 expression is critical for hematopoietic regeneration after chemotherapy[J].Cell Commun Adhes,2005,12 (5):307-317.
[12]Krenacet T, Rosendaal M. Connexin43 gap junctions in normal, regenerating, and cultured mouse bone marrow and in human leukemias: their possible involvement in blood formation[J]. Am J Pathol, 1998,152(4): 993-1004.
[13]Ito K, Hirao A,Arai F, et al. Reactive oxygen species act throupgh p38 MAPK to limit the lifespan of hematopoietic stem cells[J]. Nat Med, 2006,12(5):446-451.
[14]Juntilla MM, Patil VD,Calamito M, et al. AKT1 and AKT2 maintain hematopoietic stem cell function by regulating reactive oxygen species[J].Blood, 2010,115(20):4030-4038.
[15]Kinder M, Wei C, Shelat SG, et al. Hematopoietic stem cell function requires 12/15-lipoxygenase-dependent fatty acid metabolism[J]. Blood, 2010,115(20):5012-5022.
[16]Owusu-Ansah E, Banerjee U. Reactive oxygen species prime Drosophila hematopoietic progenitors for differentiation[J]. Nature, 2009, 461(7263):537-541.
[17]Lewandowski D, Barroca V, Duconqé F,et al. In vivo cellular imaging pinpoints the role of reactive oxygen species in the early steps of adult hematopoietic reconstitution[J]. Blood, 2010, 115(3):443-452.
[18]Wang Y, Liu L, Pazhanisamy SK, et al. Total body irradiation causes residual bonr marrow injury by induction of persisrtent oxidative stress in murine hematopoietic stem cells[J]. Free Radic Biol Med, 2010, 48(2):348-356.
[19]Gonzalez-Nieto D, Li L, Kohler A, et al. Connexin-43 in the osteogenic BM niche regulates its cellular composition and the bidirectional traffic of hematopoietic stem cells and progenitors[J]. Blood, 2012,119(22): 5144-5154.
[20]Cancelas JA, Koevoet WL,de Koning AE,et al. Connexin43 gap junctions are involved in multiconnexin-expressing stromal support of hematopoietic progenitors and stem cells[J]. Blood, 2000,96(2):498-505. |