[1]Mathur R, Joels L, Akande A, et al. The prevention of ovarian hyperstimulation syndrome [J]. Br J Obstet Gynaecol, 1996, 103(8):740-746.
[2]Ge HS, Huang XF, Zhang W, et al. Exposure to human chorionic gonadotropin during in vitro maturation does not improve the maturation rate and developmental potential of immature oocytes fromápatients with polycystic ovary syndrome [J]. Fertil steril, 2008, 89(1):98-103.
[3]Bentov Y, Esfandiari N, Burstein E, et al. The use of mitochondrial nutrients to improve the outcome of infertility treatment in older patients[J]. Fertil steril,2010, 93(1):272-275.[4]Van Blerkom J, Davis PW, Lee J.Fertilization and early embryolgoy: ATP content of human oocytes and developmental potential and outcome after in-vitrofertilization and embryo transfer[J]. Hum Reprod, 1995, 10(2):415-424.
[5]Liu Sh, Li Y, Gao X, et al. Chang of the mitochondrial distribution in human oocytes before and after in vitro maturation[J]. Acta Anatomica Sinica,2007,38(5):593-596.(in Chinese)
刘姗, 李媛, 高选, 等. 人卵母细胞体外成熟前后线粒体分布的变化[J]. 解剖学报,2007,38(5):593-596.
[6]Van Blerkom J, Davis P. Mitochondrial signaling and fertilization [J]. Mol Hum Reprod, 2007, 13(11):759-770.
[7]Santos TA, El Shourbagy S, St John JC. Mitochondrial content reflects oocyte variability and fertilization outcome[J]. Fertil steril, 2006, 85(3):584-591.
[8]Torner H, Brussow KP, Alm H, et al. Mitochondrial aggregation patterns and activity in porcine oocytes and apoptosis in surrounding cumulus cells depends on the stage of pre-ovulatory maturation[J]. Theriogenology, 2004, 61(9):1675-1689.
[9]Acton B, Jurisicova A, Jurisica I, et al. Alterations in mitochondrial membrane potential during preimplantation stages of mouse and human embryo development[J]. Mol Hum Reprod, 2004, 10(1):23-32.
[10]Combelles C, Cekleniak N, Racowsky C, et al. Assessment of nuclear and cytoplasmic maturation in in-vitro matured human oocytes[J]. Hum Reprod, 2002, 17(4):1006-1016.
[11]Wilding M, Coppola G, Dale B, et al. Mitochondria and human preimplantation embryo development[J]. Reproduction ,2009, 137(4):619-624.
[12]El Shourbagy SH, Spikings EC, Freitas M, et al. Mitochondria directly influence fertilisation outcome in the pig[J]. Reproduction, 2006, 131(2):233-245.
[13]Stojkovic M, Machado SA, Stojkovic P, et al. Mitochondrial distribution and adenosine triphosphate content of bovine oocytes before and after in vitro maturation: correlation with morphological criteria and developmental capacity after in vitro fertilization and culture[J]. Biol Reprod,2001, 64(3):904-909.
[14]Motta PM, Nottola SA, Makabe S, et al. Mitochondrial morphology in human fetal and adult female germ cells[J]. Hum Reprod, 2000, 15(suppl 2):129-147.
[15]Zeng H, Yeung WSB, Cheung MPL, et al. In vitro-matured rat oocytes have low mitochondrial deoxyribonucleic acid and adenosine triphosphate contents and have abnormal mitochondrial redistribution[J]. Fertil Steril, 2009, 91(3):900-907.
[16]Zeng H, Ren Z, Yeung WSB, et al. Low mitochondrial DNA and ATP contents contribute to the absence of birefringent spindle imaged with PolScope in in vitromatured human oocytes[J]. Hum Reprod,2007, 22(6):1681.
[17]Lee ST, Oh ST, Lee EJ, et al. Adenosine triphosphate synthesis, mitochondrial number and activity, and pyruvate uptake in oocytes after gonadotropin injections[J]. Fertil Steril, 2006, 86(4): 1164-1169.
[18]Yu T, Jhun BS, Yoon Y. High-glucose stimulation increases reactive oxygen species production through the calcium and mitogen-activated protein kinase-mediated activation of mitochondrial fission[J]. Antioxid Redox Signal, 2011, 14(3):425-437.
[19]Lopes A, Lane M, Thompson J. Oxygen consumption and ROS production are increased at the time of fertilization and cell cleavage in bovine zygotes[J]. Hum Reprod, 2010, 25(11):2762-2773.
[20]Miyamoto K, Sato EF, Kasahara E, et al. Effect of oxidative stress during repeated ovulation on the structure and functions of the ovary, oocytes, and their mitochondria[J]. Free Radic Biol Med, 2010, 49(4):674-681.
|